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This response is defective in gustducin -null mice, which have an impaired GLP-1 response to luminal glucose (152)

10.1016/s0928-4257(97)89478-8 6 HuangT.ZhangK.SunL.XueX.ZhangC.ShuZ.et al (2015)

Cellular iron homeostasis is regulated at the post-transcriptional level through iron-responsive element (IRE)iron regulatory protein (IRP) interactions, which control the translation of mRNAs encoding ferritin, FPN, TfR1, and DMT1.Comprising ferrous iron bound by low-affinity iron chelators, NTBI is a direct consequence of oxidative stress in the circulation and tissue iron loading [30]

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This is compared to 3.1% of patients without such ulcers