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doi: 10.1007/s12291-014-0446-0

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Mnzel T, Gori T, Keaney JF Jr, Maack C, Daiber A (2015) Pathophysiological role of oxidative stress in systolic and diastolic heart failure and its therapeutic implications

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Additionally, Tat-conditioned microglial EVs enriched for NLRP3 convey pro-inflammatory signals and contribute to synaptodendritic injury, further impairing neuronal structure and function [58]

Therefore, while the GM represents a key modulator of bone homeostasis, its precise function and its contribution are likely conditioned by the predominant underlying etiology of bone loss and the specific skeletal site examined