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These results suggest that Sema can counteract the TAC-induced activation of the PI3K/AKT/Creb5/NR4a1 signaling pathway and reduce NR4a1 nuclear-mitochondrial translocation to ameliorate TAC-induced cardiac remodeling

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Semaglutide is widely studied in metabolic, biochemical and receptor-signaling research, including investigations involving glucose-dependent insulin signaling, glucagon regulation, appetite-related pathways, gastric emptying and energy metabolism