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N-Acetylcysteine Suppresses Microglial Inflammation and Induces Mortality Dose-Dependently via Tumor Necrosis Factor- Signaling

Even so, O'Donnell does not see this as an existential blow

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This discovery is significant not only for understanding the diseases mechanisms but also for exploring new therapeutic approaches that target DPP4 activity to restore immune balance and improve treatment outcomes for SLE patients