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Therefore, only the backbone is modelled in the final pdb file

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This dysregulation has been closely linked to the pathogenesis of RA (95)

Additionally, it could be hypothesized that cellular proteins with thiol-disulfide active sites, such as GSR, could contribute not only to reduce GSSG levels and to the normalization of the GSH/GSSG ratio, but also to decrease AX availability by a thiol-mediated mechanism