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These data support an effect of IL-13 to decrease total and reduced GSH through high 15LO1, PEBP1, and GPX4 activity, which is partially compensated by (a) increased expression/activity of SLC7A11 to enhance new intracellular GSH production and (b) increased export of GSSG

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At the same time, it has a similar function to GPX7, which can increase the PDI activity of ER redox protein 1 (ERO1), promote the oxidative folding of endoplasmic reticulum proteins, and reduce oxidative stress (Yoboue et al., 2017)

Specifically, GPER may promote the proliferation of thyroid cancer cells by regulating the expression of cell cycle-related genes [202]

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Our findings also underscore the crucial role of central redox mechanisms, particularly in the mPFC, in mediating the pathophysiological effects of THC