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While the IH-dependent pro-oxidant state appears to negatively regulate both critical periods, cell-autonomous HIF1a signaling during the first critical period of adult neurogenesis appears to orchestrate adaptations that enhance the neuronal generation following termination of IH

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For instance, empirical evidence shows that rat hepatocytes begin ROS production after the first 30 minutes of DEM exposition (5 mM), growing linearly for all the remaining time, whereas the increase in products of lipid peroxidation (TBARS) starts only from the first hour of exposure (Tirmenstein et al

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