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[7] Not all infants with low free carnitine are affected with SPCD

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However, the mutant strains expressing non-phosphorylatable- vadK:vadK His422Ala , phosphomimetic vadK:vadK His422Asp or a non-functional ATP-binding site vadK:vadK Arg106Lys were unable to grow (Appendix Fig

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The more clinically relevant question is whether controlled glucagon receptor activation, when paired with GLP-1 and/or GIP receptor agonism, can shift glucagons metabolic impact from primarily hyperglycemic toward thermogenic, lipolytic, and hepatometabolic benefit