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Temozolomide drives ferroptosis via a dmt1-dependent pathway in glioblastoma cells
Given this fact, as well as homocystines proposed mechanisms of increasing endothelial dysfunction through oxidative damage, increased platelet coagulation and vascular smooth muscle cell proliferation, we recommend that homocysteine should be assessed and treated when levels rise, to reach a level of below 7 mol/L
However, conventional oral glutathione is poorly absorbed and rapidly degraded during digestion
These coefficients are provided in Supplementary Data 10
