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GSH depletion led to defects in actin-based motility and cell-to-cell spread

Regulating macrophage ferroptosis and macrophage-derived foam cells has emerged as a novel therapeutic target for coronary atherosclerotic heart disease

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This dual failure of excessive mitochondrial fragmentation coupled with deficient clearance results in the toxic accumulation of damaged, ROS-generating mitochondria

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Antioxidant activity of IP 6 is seen to be especially relevant for the xanthine/xanthine oxidase system, which generates H 2 O 2 by consecutive hypoxanthine to xanthine and xanthine to uric acid oxidation reactions [126]