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In EAE mice, adoptive transfer of itaconate-treated, Th17-polarized T cells significantly ameliorated disease severity (173), highlighting the potential of targeting metabolic pathways to modulate the Th17/Treg balance in autoimmune neuroinflammation

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Another recent report postulated that STING-induced LC3 lipidation happens independent of WIPI2 at perinuclear single membrane vesicles, rather than double-membrane autophagosomes, and requires both the V-ATPase as well as the WD40 domain of ATG16L1 required for interaction with V-ATPase (161, 162)