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The mechanism by which thiol-binding metals exert a toxic activity on cells and organisms is in general terms described as the consequence of interaction with and inhibition of essential thiol groups of enzymes and proteins, although the systemic toxicity of the metals is very different [2]

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In DSS and TNBS induced colitis models, orally administered KPV has consistently reduced disease activity scores, lowered inflammatory cytokine levels, decreased mucosal damage, and helped restore gut barrier function

PMID 17084875

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To ensure that allosteric sites are in sufficient distance from the catalytic site, we have calculated distances between the nearest atoms as well as C atoms of nearest H-bond forming residues of the allosteric and catalytic sites