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doi:10.1128/AAC.00779-06 78

FIGURE 10 Oxidative stress, associated with high dose MTX, triggers a systemic inflammatory response where proinflammatory cytokines further contribute to tissue damage ( During stressful conditions, such as MTX-induced oxidative damage, unresolved ER stress leads to the accumulation of unfolded proteins promoting apoptosis (Figure 1)

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No mutations of receptors or other elements of the signaling mechanisms have been identified in T1D so far