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Liver IRI is characterized by two interlinked phases: ischemia-induced hepatocyte injury, leading to the release of damage-associated molecular patterns, and subsequent recruitment of immune cells, initiating an inflammatory cascade that exacerbates hepatocyte damage

To determine the relevance of these amino acid residues for the interaction, we generated SNIP1 mutants K265G, K301G and R365G and tested their binding to P-SF3B1 in an in vitro binding assay (Supplementary Fig

It has shown superior efficacy compared to some other diabetes medications

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