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The potential to counteract these effects has been demonstrated with IL-1 receptor antagonists, which have been shown to improve glucose tolerance and enhance insulin production [571]

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Further investigation on signal transduction pathways among the three major pancreatic endocrine cell types demonstrated that the probability of interaction between key regulators of glucagon secretion, such as ligand-receptor pairs Sst-Sstr2 , Sst-Sstr3 , Ins1-Insr , and Ins2-Insr , were significantly reduced in GCGR-KO cells (Fig

CHAC1/MGC4504 is a novel proapoptotic component of the unfolded protein response, downstream of the ATF4-ATF3-CHOP cascade

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