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PLoS ONE 7 , e37613 (2012)
Cisplatin accumulates persistently in the mitochondria of energy metabolism-related organs (skeletal muscle, liver), forms platinum-mtDNA adducts to disrupt mitochondrial structure and function, and causes oxidative phosphorylation impairment and insufficient ATP production, ultimately driving persistent fatigue (Huot et al
The unique monomer structure is speculated to allow the reduction of more complex lipid peroxides, although this has only been proven for GPX4
It serves as a primary internal defense mechanism, helping to maintain cellular health and supporting the immune system
Additionally, around 12% of oxygen used in mitochondrial metabolism produces free radicals, which can lead to oxidative stress and even DNA damage [2]
Tilkian S, Conover M, Tilkian AG
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