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The inactivity of the lipid repair enzyme GPX4 may result in elevated levels of lipid-based ROS, hence triggering ferroptosis [56]

The mentioned effect was observed in vitro or in animal models

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As these were already lower in the mutants compared to the WT this led to stronger phenotypes regarding root length and total biomass production

A lipidomic study of phospholipid classes and species in human synovial fluid