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FIGURE 9 The core of this toxicity mechanism lies in the depletion of the labile Fe 2+ pool (Figure 5B)without altering total cellular iron (Figure 5A)which triggers a compensatory iron-starvation response (IREB2, TfR1, Ferritin

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The activation of microglia is typically bifurcated into two distinct phenotypes: the M1 phenotype, which is linked to pro-inflammatory activities and neurotoxic responses, and the M2 phenotype, which is engaged in tissue repair and neuroprotective processes

The pro-inflammatory effects of miR-155 promote liver fibrosis and alcohol-induced steatohepatitis