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These doses of methanolic extract of Shatavari decreased corticosterone as well as norepinephrine levels and showed anxiolytic activity by modulating GABA and serotonergic-mediated pathways further confirming its therapeutic potential for the treatment of anxiety disorders[53]

The proposed mechanisms are multifaceted

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Central Nervous System Effects : Perhaps most intriguingly, research has revealed that the anti-inflammatory effects of GLP-1 receptor agonists require GLP-1 receptors in the central nervous system, not just in blood or endothelial cells

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