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falciparum has no identified Nrf2 orthologue and so likely lacks a KEAP1-Nrf2 mediated antioxidant response, which suggests that the SFN-GSH conjugate should only cause oxidative stress in parasites

Moreover, increased renal glucose production is a possible contributor to the development of hyperglycemia in patients with insulin resistance and MetS (Legouis et al., 2022)

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Animal model research demonstrated that depleting GSH via inhibition of GCL triggered a cascade of lipid peroxidation and cell death in mesencephalic cultures, identifying oxidative stress from glutathione insufficiency as a likely initiating mechanism in neurodegeneration

Similar content being viewed by others Background Recombinant DNA and protein technologies have taken a leading role in many forms of research over the past 40 years [1]

The binding energies were all lower than 5.0 kcal/mol