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5d) therefore suggests a less efficient interaction between E170X mutants and HEDS-derived GSSEtOH in accordance with the scaffold model and the hypothesis that GSSEtOH formation is catalysed by ScGrx7

J Allergy Clin Immunol Pract 8(9S):S16S116 Lin D, Li WK, Rieder MJ (2007) Cotrimoxazole for prophylaxis or treatment of opportunistic infections of HIV/AIDS in patients with previous history of hypersensitivity to cotrimoxazole

One such mechanism, which provides immune privilege and prevents NK-cell-mediated cytotoxicity, is the intracellular localization within melanoma cells of the ligand that typically activates NKD2D receptors on NK-cells [31]

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doi:10.1056/NEJMoa2107519 192