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In the classical ferroptosis regulatory pathway, GSH is depleted by erastin or other system Xc- inhibitors, which reduces cysteine synthesis and leads to GSH synthesis disorder [2, 72, 73]

have revealed that the activating molecule in beclin1-regulated autophagy (Ambra1) localized to mitochondria may also contribute to autophagosome formation by interacting with Beclin1 [285]

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