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The pathogenetic mechanism of HS relies on loss of membrane surface and resultant decreased membrane surface area to volume ratio leading to increased cell sphericity and decreased cellular deformability

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Nonetheless, despite these limitations, our study offers significant insights into the direct impact of metformin on intestinal glucose metabolism, highlighting the role of TXNIP downregulation and GLUT1 upregulation as a key mechanism of its glucose-lowering effect

FOXO4-DRI demonstrates preferential targeting of high-SASP senescent cells, showing an 11.73-fold selectivity for senescent versus normal cells in controlled studies

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