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Human clinical trials have not been completed, meaning efficacy and safety in people is not established through rigorous research

In animal studies spanning three decades, no lethal dose has been identified

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This outcome results from combined mechanisms of increased fatty acid oxidation and decreased lipogenesis

Although breast cancer cell lines exhibit increased resilience to physiologically relevant shear stress compared to normal epithelial cells [223], lamin A/C knockdown exacerbates shear stress-mediated tumor cell death [223], suggesting that elevated lamin A/C levels could aid breast CTCs in withstanding shear forces in the circulation