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Furthermore, a large number of genetic variants have been described in the UGT genes that are due to mutations in both the coding and regulatory regions of the genes[4853]

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Anita Gupta of the Johns Hopkins School of Medicine, who served on FDA's compounding committee until earlier this year

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Treatment with PT2385 resulted in significant tumor regression and prolonged survival in preclinical models of VHL-deficient ccRCC, without notable systemic toxicity (188)