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In contrast to chronic treatment with intact hGH, chronic treatment with AOD9604 showed no adverse effect on insulin sensitivity of the animals, as demonstrated with euglycemic clamp techniques Another study published in 2001 found the same thing, yet the results were very different for lean rats versus obese rats: Both hGH and AOD significantly reduced body weight gain in obese mice
In adipose tissue, inhibition of NNMT suppresses de novo lipogenesis, normalizes fat content, and improves insulin sensitivity 2-3
The therapeutic mechanism may be associated with accelerated granulation tissue formation, reepithelialization, dermal remodeling, and collagen deposition through ERK1/2 signaling pathway
The dosages and protocols discussed here are general references from published studies and experimental reports, not individualized prescriptions
[7] This dual route is the mechanistic feature that most clearly separates survodutide from GLP-1 mono-agonists in the liver-disease setting, where reducing hepatic fat is the entry point to slowing inflammation and, eventually, fibrosis
