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Specifically, this includes the generation of mtROS, alterations in mitochondrial morphology and function, increased calcium ion uptake, and the regulation of specific genes, all of which collectively contribute to ferroptosis

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Transcriptomics and biochemical evidence of trigonelline ameliorating learning and memory decline in the senescence-accelerated mouse prone 8 (SAMP8) model by suppressing proinflammatory cytokines and elevating neurotransmitter release