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Therefore, the problem that the agents successful in pre-clinical experimental autoimmune encephalitis trials, were either less effective in patients, worsened disease or caused unexpected, severe adverse events (60), may be at least partly solved with the more suited cuprizone model that reflects a toxic experimental model (12)

This approach revealed that reduced periplasmic glutathione levels indeed resulted in significantly lower PhoA activity, in line with the more reduced roGFP-iL redox state in gshA

D-gal increased the levels of the pro-inflammatory cytokines IL-2 and IFN-

In addition, basal STING levels seem to be kept under control by the ER-associated degradation (ERAD) system

Across the seven substances, committee members consistently returned to four questions: Is the bulk drug substance sufficiently identified and characterized

High uptake of [2- 11 C]acetyl-L-carnitine into the brain: a PET study Biochem Biophys Res Commun 1997