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Previous research indicates that individual mitochondrial respiratory complexes are mutually linked, and compensatory responses can be engaged to manage changes in substrate availability, metabolic flux, and ETC component function to meet bioenergetic demands 13
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C.RinkerD
20 February 2024
The proposed mechanismsenhanced hepatic fat metabolism and increased lipolysislack robust human evidence
identified two genes, charged multivesicular body protein 1a (CHMP1A) and dipeptidase 1 (DPEP1), associated with kidney disease that could regulate ferroptosis by altering cellular iron transport [141]
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