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Inhibition of key regulators of 2D cell locomotion such as actin polymerization, cell-matrix adhesion, and myosin contractility fail to block confined migration of MDA-MB-231 breast cancer cells [153], suggesting the existence of alternative mechanisms facilitating their movement

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Moreover glutaminolysis-mediated mTORC1 activation required prolyl hydroxylase (PHD) enzymatic activity in a HIF -independent manner [89]

Since cancer cells typically rely on glucose as their primary nutrient for energy production, restricting glucose intake is a promising therapeutic approach

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