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Inclusion criteria encompassed all studies employing in vitro models (e.g., fibroblast proliferation, keratinocyte migration, collagen synthesis assays), in vivo studies using animal models for wound healing assessments, clinical trials evaluating the efficacy and safety of tripeptides in human subjects, and studies utilizing molecular, biochemical, or histological analyses to determine the mechanisms of tripeptides in wound recovery

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Access this article Similar content being viewed by others Data availability The 16S rDNA sequence of SP4 was submitted to GenBank with accession number KT306966

FIN56, as a ferroptosis inducer, can inhibit the integration of Sec on the GPX4 catalytic subunit, thereby reducing the expression of GPX4s antioxidant activity ( 3.1.5 Glutamine (Gln) metabolism pathway The Gln metabolism pathway is another important pathway involved in the regulation of ferroptosis