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CYP2D6 may also contribute to the formation of NAPQI, albeit to a lesser extent than other P450 isozymes, and its activity may contribute to paracetamol toxicity, in particular, in extensive and ultrarapid metabolizers and when paracetamol is taken at very large doses.[18] The metabolism of paracetamol is an excellent example of toxication, because the metabolite NAPQI is primarily responsible for toxicity rather than paracetamol itself

and it can contribute to anxiety as well

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I followed her recommendation, and I have noticed a total transformation

doi:10.3748/wjg.v20.i22.6725

Standard receptor pharmacology principle, applied to incretin signaling