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Fasting glucose, insulin levels, glycated hemoglobin, and lipid profiles all improved significantly across dose groups

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doi:10.1111/cts.13109

Cell culture studies using adipocytes have shown that NNMT overexpression promotes lipid accumulation and insulin resistance, while NNMT knockdown or inhibition increases NAD+ levels, activates SIRT1, enhances mitochondrial respiration, promotes expression of thermogenic genes including UCP1, and drives white adipocyte browning toward a beige phenotype

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The increased NAD+ availability activates SIRT1, a protein that plays a crucial role in regulating how your body handles blood sugar