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Severity and Profile of Autism: The specific pattern of strengths and difficulties shapes therapy priorities

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Two independent mechanisms = a potentially synergistic combination

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Individuals with TPMT deficiency should avoid thiopurine treatment or, if deemed necessary, start with 125 pmol/8 10 8 RBC had a significantly higher median infliximab trough level than those with 6-TGN levels A (rs1127354), which is associated with low enzyme activity ( Thiopurine Hypermethylation and Therapeutic Resistance Thiopurine compounds are associated with a high rate of non-response, which occurs in approximately 50% of patients given the drugs ( The 6-TGN under-production in a subgroup of IBD patients is largely believed to be due to thiopurine hypermethylation ( shunters ) whereby thiopurine compounds are preferentially metabolized into methyl-mercaptopurine (meMP) and meMPR ( Figure 1 ), resulting in inadequate 6-TGN levels and therefore treatment resistance ( 8 RBC has been found to cause hepatotoxicity and other thiopurine-induced adverse drug reactions ( The role of TPMT in inducing thiopurine hypermethylation has been a subject of great interest

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