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Conda-Sheridan, M., & Krishnaiah, M

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The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

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The tripeptide's copper-binding stoichiometry (1:1 Cu2+ to GHK) is studied using spectroscopic methods

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