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Breindahl T, et al

(2022) reported JUN, an oncogenic transcription factor, as a regulator of glycolytic metabolism in AML, since it increases the expression of hexokinase I (HKI) and HKII, glucose phosphate isomerase (GPI), phosphofructo-1-kinase (PFKP), aldolase A (ALDOA), glyceraldehyde-3-phosphate dehydrogenase (GAPDH), phosphoglycerate kinase (PGK1), enolase 1 (ENO1), and PKM

[DOI] [PMC free article] [PubMed] [Google Scholar] 47.Masson W., Lobo M., Nogueira J.P., Rodriguez-Granillo A.M., Barbagelata L.E., Siniawski D

Personalized Genetics and Individual Response to Semaglutide The PlexusDx Precision Peptide Genetic Test analyzes GLP1R (rs6923761), GIPR (rs1800437), FTO (rs9939609), and MC4R (rs17782313) variants that relate to GLP-1 and related signaling pathways

Conclusions The whole range of primary genetic astrocytopathies is yet to be fully characterised

24 and 48 h later, viral supernatant was collected and filtered through a 0.45 m filter