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GLP-1 medications mimic a hormone the body naturally produces after eating

doi:10.1038/nn1405

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Studies done in drug-nave animals revealed that activation GLP-1-producing neurons in the NTS are not necessary for the anorexic effects of systemic GLP-1R agonists and that concurrent NTS GLP-1-producing circuit activation suppresses feeding more potently than GLP-1R agonism alone 71

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