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This ethanol metabolism in LSECs leads to a decrease in nitric oxide (NO) production, largely due to alterations in the acetylation of heat shock protein 90 (Hsp90) and its interaction with endothelial nitric oxide synthase.24 Importantly, studies suggest that blocking Hsp90 acetylation may restore NO production and improve LSEC function.24 In addition to ethanol metabolism, LSECs are implicated in the progression of fibrosis and inflammation in liver diseases

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Liposomes can be readily functionalized for active targeting, thereby enhancing their ability to cross the BBB and improve pharmacokinetic profiles [70]

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