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With no clinically approved treatments or diagnostic markers for these conditions, there is an urgent need to define the molecular underpinnings

Because cysteine availability is a limiting factor for glutathione synthesis, treatment with N-acetylcysteine may increase glutathione levels and thereby counter oxidative stress, promote redox -regulated cell signaling, and improve immune responses

Y., McBride, S

This process facilitates endosomal uptake of nucleic acids, shielding them from nuclease degradation and subsequently inducing TLR7- and TLR9-mediated production of IFN-Is and TNF- in diverse immune cells, such as B cells and pDCs

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