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When either nutrient is lacking, DNA synthesis is disrupted, leading to impaired cell division and the production of abnormal red blood cells

Studies suggest that the VEGF/VEGFR-2 pathway may be particularly relevant, as its activation seems to trigger intracellular signaling cascades that potentially influence endothelial cell migration, proliferation, and survival

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Studies on cellular energy balance and fat metabolism pathways

The implications of this in people with Down syndrome need additional study

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